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TNF- induces inflammation by preparing leukocytes to synthesize oxidants, triggering prostaglandin and other inflammatory mediators, and causing adhesion molecules to be produced, attracting leukocytes to tissue injury sites [55]

Critically, these decidualization and matrix remodeling defects do not occur in isolation: they frequently co-occur with immune dysregulation, suggesting that RIF may represent a syndrome of coordinated endometrial dysfunction rather than a single-pathway failure [287, 297]

The fact that GHK-Cu not only stimulates the production of dermal components, but also regulates their breakdown suggests that it should be used with caution

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