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To me this is interesting for three reasons: Diagnosis - staining biopsy tissue for ApoE may simplify diagnosis of DDD Disease mechanism - the authors suggest that dense deposits may form as ApoE binds to proteoglycans in the GBM and scavenges C5-9, particularly in the face of increased complement activity Treatment - are terminal pathway treatments such as C5 inhibitors going to evolve into key tools for DDD in particular
What is the niacinamide in Empower tirzepatide for
Gao Q, et al
IGF-1 haploinsufficiency causes age-related chronic cochlear inflammation and increases noise-induced hearing loss
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