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Of note, neuroinflammation has furthermore been implicated in the pathophysiology of depression [87], whereas increased inflammatory markers, such as C-reactive protein (CRP) or interleukin-2 (IL-2), have been specifically described to be more associated with the atypical subtype of depression [69]

It was noted that the effects of UK-5099 treatment on GSH reduction and replication stress were less in 32D cells (Fig

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Li et al., 2014)