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256 Conclusion and Future Directions TMAO may drive the cross-talk between inflammatory response and oxidative stress by mediating cell death (including apoptosis, pyroptosis, autophagy, and ferroptosis, etc) and then induce pathological processes such as foam cell activation, massive secretion of cytokines and adhesion molecules, overaccumulation of ROS, enhanced platelet reactivity and abnormal vascular tone regulation

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It declines naturally with age and stress

What is GLP-1, and why does the microbiome come into it