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Intriguingly, the interactions of N-terminal histidine residues of the prion protein (PrP) mainly with Cu 2+ sequestration and accumulation in diseased brain (T 2+ at neutral pH (7.4) ( 2+ is not necessarily pathogenic since it has also been found to produce neuroprotection due to the cis interaction with the C terminal of PrP
John B Pritchard critical comments of this manuscript
Since the accumulation of uremic toxins in CKD patients engenders various neurological complications, treatments that target the significant reduction of these metabolites not only significantly delay CKD progression but also greatly improve cognitive function in these patients (Faucher et al., 2023)
Thusly, ferroptosis is considered to be an important pathway leading to IS
Leitch JM, Li CX, Baron JA, Matthews LM, Cao X, Hart PJ, Culotta VC
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