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Preclinical studies likewise suggest a favorable safety profile [12, 13]
In addition, deletion of GLS1 genes from Myc-driven liver tumors upregulates several metabolic compensatory pathways, including glycolysis and aminotransferases 111
Peptides in general are poor substrates and poor inhibitors of cytochrome P450 enzymes because they are typically degraded by proteases rather than hepatic oxidative metabolism [7]
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