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By inhibiting URAT1, arhalofenate promotes the excretion of uric acid in the urine, reducing serum uric acid levels
This is evidenced by the negligible affinity of amylin or pramlintide for the isolated CTR ECD 19,20 , and by loss of affinity and/or potency of P32Y substituted CT peptides at the CTR 18,20
It binds to heavy metals, environmental pollutants, and metabolic waste, making them water-soluble so the body can flush them out
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