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GLP-1 remains the backbone, but layering complementary signals from GIP, glucagon, amylin and PYY allows for unprecedented degrees of weight reduction and metabolic restoration, as summarised for comparison in Table 1
Its connection to mitochondria makes it an unparalleled tool for that specific question
In animal models, nor-ursodeoxycholic acid, a synthetic bile acid homolog, has been demonstrated to decrease liver enzymes, fibrosis, and inflammation
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