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The dissociation/relaxation of nCRP into nnCRP occurs on necrotic, apoptotic, and ischemic cells, membranes of activated platelets, monocytes, and endothelial cells, and on the surface of microparticles, via phosphorylcholine binding and seems to be, as mCRP, proinflammatory (194196)
Growing evidence suggests that numerous cancers, including hematopoietic tumors, may be driven by a small subset of cells known as tumor-initiating cells (TICs) [15]
Furthermore, emerging evidence indicates that TJ protein alterations may also contribute to BBB dysfunction in psychiatric conditions such as bipolar disorder and schizophrenia, where they are associated with cognitive impairments and neuroinflammation [286]
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