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Animal models show that GLP-1 drugs improve memory, reduce neural apoptosis, and increase synaptic density

The glucagon receptor agonism is particularly notable because glucagon increases energy expenditure, promotes hepatic fatty acid oxidation and ketogenesis, and enhances lipolysis primarily in adipose tissue effects that may amplify overall weight reduction beyond what GLP-1 or GIP activation alone can achieve

Prevent Gallbladder Issues Gallbladder events (1.4% in trials) primarily occur during rapid weight loss phase (months 4-6)

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In patients with T2DM and hepatic steatosis, Cuthbertson et al