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injection of exenatide dampened cocaine-driven septal dopamine elevation, mediated by postsynaptic GLP-1R signaling that retrogradely regulates presynaptic dopamine transporter function [106]

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3 shows univariate regression analysis for GLP-1 secretory responses after oral glucose ingestion, from which responses appear to be predicted by baseline characteristics such as age, body weight (obesity), fasting NEFA concentrations and fasting glucagon concentrations