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The authors found that BPDB-based MOFs exhibited GSH concentration-dependent NO release (Figure 2G), and four molecules of NO can be released from each BPDB molecule provoked by two molecules of GSH (Figure 2H)

In the rabbit bone defect study, BPC-157 treatment produced complete bony continuity across the defect site within six weeks in treated animals, while all control animals remained unhealed

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