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Mir-135 suppresses glycolysis and promotes pancreatic cancer cell adaptation to metabolic stress by targeting phosphofructokinase-1

OxPhos is achieved in the presence of saturating ADP levels and the sequential addition of different mitochondrial substrates, including pyruvate, malate, glutamate (N-linked, through complex I) and succinate (S-linked, through complex II)

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pastoris (GAP), and a galactose-based GAL1 promoter of S