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Research indicates that HGF activation provides neuroprotective effects in cerebral ischemia, spinal cord injuries, and traumatic brain damage by promoting neuronal survival, stimulating angiogenesis, and reducing apoptosis

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Through integrated proteomic and metabolomic analyses, we identified a high-inflammation, high-oxidation, low-metabolism triad in the hippocampus of CLP-induced SAE mice, providing novel insights into mechanisms underlying persistent cognitive deficits in SAE survivors

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