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B.LyonsweilerJ.KooninE
Stalled ribosomes and stop codon read-through can both activate cellular stress pathways (such as ribosome quality control complexes and the unfolded protein response) and induce oxidative stress (ROS) that downregulate translation and regulate cellular survival 114 (Fig
15 Natural compounds like fisetin and quercetin have credible early dataand may offer real benefits while pharmaceutical-grade precision senolytics continue development
At the molecular level, the PINK1/Parkin-dependent pathway for mitochondrial autophagy is a key mechanism [112]
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