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In hypertension models, GLP-1 RAs exert beneficial effects through multiple mechanisms including vascular vasodilation, diuresis, decreased sympathetic activity in the central nervous system, and reduced blood pressure variability (BPV) [15]
GPX4, the central regulator that drives ferroptosis resistance, was analyzed in PyMT cells after treatment with RSL3 (Fig
PMID: 25596009
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