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The advantage is that those risks are more predictable and more manageable than the systemic disruption caused by broad-acting compounds

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These breakthroughs often lead to a renewed sense of confidence and motivation, encouraging patients to continue their health journeys with enthusiasm

Studies have shown that in the HF mouse of myocardial infarction models, the depletion of neutrophils through antibody-mediated or genetic methods can halt the progression of HF, especially the left ventricular remodeling and fibrosis progression in the middle and late stages of HF (135), further indicating that neutrophils are a potential therapeutic target for lowering myocardial damage and preventing HF (159, 160)