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GlyNAC (glycine and N-acetylcysteine) supplementation in mice increases length of life by correcting glutathione deficiency, oxidative stress, mitochondrial dysfunction, abnormalities in mitophagy and nutrient sensing, and genomic damage
This, in turn, contributes to a reduction in antioxidant capacity
Here, we added the scFv of the CD3 antibody (patent US9650446B2) to the N-terminus of FcRIIIa and used the Her2 antibody, trastuzumab, as a representative IgG1
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