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The GLP-1 RA and Its Relationship with Brain Insulin Resistance Brain IR can be defined as the failure of brain cells to respond to insulin, and the lack of response may be due to the downregulation of insulin receptors, an inability of insulin receptors to bind insulin, or faulty activation of the insulin signaling cascade

Cell Metab 27

Sources: Short KR, et al

These findings indicate that, under AD conditions, astrocytes and neurons exhibit distinct autophagic responses

Abstract Introduction: The surge in prescribing glucagon-like peptide-1 receptor agonists (GLP-1 RAs) for diabetes and weight management highlights a pressing need to characterize their associated risks