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Therefore, in response to iron accumulation and high levels of ROS within AT, certain anti-inflammatory immune cells may also be affected by the ferroptosis process, resulting in their inability to neutralize the inflammatory response caused by pro-inflammatory cytokines in a timely manner

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Since we previously found that ser/gly rewired tumors produce and secrete high levels of pathway metabolites into the circulation, we hypothesized that metabolomic analysis of blood plasma samples from patients with NSCLC could give valuable insight into ser/gly metabolic rewiring in tumor cells in response to RT [24]

There's one more early finding worth holding onto, because it comes back later and it's an odd one