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312 Lipotoxicity impairs insulin signaling, induces oxidative damage, promotes inflammation and fibrosis, 313 which is thought to be related to the progression of MAFLD patients from simple steatosis to NASH, hepatic fibrosis, and hepatocellular carcinoma
Even more importantly, BPC 157 accordingly inhibited corticosteroid-induced immunosuppression (Sikiric et al., 2003)
This reprogramming results in increased expression of M1 markers (CD80, TLR2) and the production of pro-inflammatory cytokines and chemokines, such as IL-12 and IFN, while reducing M2 markers (CD163, CD206)
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10.1111/and.13898 Andrologia
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