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This suggests that analogs may potentially influence the fibrotic process by modulating the expressions of genes involved in fibrogenesis and inflammation

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Pre-existing inborn errors of sulfur-amino-acid metabolism (e.g., homocystinuria) represent an absolute contraindication because parenteral methionine can exacerbate elevated homocysteine concentrations and associated thrombotic risk

It modulated neural activity in the ventral striatum and midbrain pathways, affecting brain areas related to metabolic signaling

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