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Ferroptosis signature in SARS-CoV-2 infection and molecular mechanisms of ferroptosis A case study of a COVID-19 patient has showed the presence of a ferroptosis signature in cardiac and renal tissues [77]
Table 1 Multi-weaponry approach involving glutathione (GSH) enhancers, nuclear factor erythroid 2 p45related factor 2 (Nrf2) activators, toll-like receptor (TLR) inhibitors/immunomodulators, C-reactive protein (CRP) level reduction, natural and immune immunoglobulin M (IgM) enhancement and immune cell function recovery against SARS-CoV-2 infection and COVID-19 disease
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As our bodies age, and depending on our environmental conditions, our natural abilities to produce many compounds like glutathione can slow down and become weaker
doi: 10.1038/s41598-021-87619-8 11
