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N-acetylcysteine augmentation in children and adolescents diagnosed with treatment-resistant obsessive-compulsive disorder: Case series
Clin Ther 1989;11:584589
Presented for research literature review only Bermes EW Jr et al
Its mechanisms include: Actin polymerization mobilizes cellular building blocks for tissue repair Cell migration accelerates fibroblast and keratinocyte movement to injury sites Angiogenesis promotes new blood vessel formation via VEGF upregulation Anti-inflammatory reduces IL-1 and TNF- at injury sites TB-500's smaller molecular size gives it excellent tissue distribution throughout the body
Islam R, Bilal H, Wang X, Zhang L
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