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leaves improved HCEC cell survival under hyperosmolar stress compared to untreated controls
1.) Improvement of all specific cell functions by normalizing the redox potential and optimizing protection against the destruction of important biomolecules and cell structures through over-oxidation, as well as protection against lipid peroxidation and others 2.) Optimization of the function of many cell enzymes by setting an optimal redox potential for the cell, partly through the formation of mixed disulfides
Early-life microbiota and nutrition: investigating the effects of fiber on maternal milk and offspring metabolic outcomes
Oxidative stress: A cause and therapeutic target of diabetic complications
Environmental and molecular mutagenesis , 58 (2), 6071
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