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Its key actions include: Stimulating glucose-dependent insulin secretion from pancreatic beta cells Inhibiting glucagon secretion , which otherwise raises blood sugar Slowing gastric emptying , which delays the absorption of glucose Promoting satiety via central nervous system signalling The role of GLP-1 was uncovered in the 1980s during research into the incretin effect the observation that oral glucose stimulated more insulin than intravenous glucose

A more likely explanation, he says, is telogen effluvium, a temporary shedding condition triggered by stress

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