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The results showed that the transient expression of SsCm1 inhibited callose deposition and the expression of pathogenesis-related genes ( NbPR1a and NbPR2 ) induced by PAMPs, including Flg22, chitin, and Sclerotinia culture filtrate elicitor (SCFE), while no significant variation was detected in the ROS burst induced by PAMPs (Supplementary Fig
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These changes contribute to regional neuronal dysfunction, memory impairment, and cognitive decline
The choice of the most appropriate therapy is up to the treating physician, based on the diagnostic criteria, the symptoms and the expected benefit
Macrophage-derived FGFR1 drives atherosclerosis through PLCgamma-mediated activation of NF-kappaB inflammatory signalling pathway
