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It helps recycle oxidized glutathione back into its active form, allowing the body to maintain higher antioxidant protection

(2018, March 12)

Similarly, in in vivo experiments, Lip-1, a ferroptosis inhibitor, decreased neuronal death and neuro-inflammation in EBI by protecting mitochondrial functions and reducing lipid peroxidation, thereby ameliorating cerebral edema and neurological deficits after SAH ( 3 The Mechanism of Ferroptosis Studies have indicated that ferroptosis may be induced by different physiological conditions and pathological stress

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