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Extensive research has unequivocally highlighted the pivotal role of Th17 cells in the pathogenesis of UC (Fitzpatrick et al., 2020
Solution stability must follow a protocol-specific validated window
Indirect lesion area was determined by subtracting the intact area of the ipsilateral hemisphere from the contralateral hemisphere
Its biological output is dynamically tuned by therapeutic modality, microenvironmental redox conditions, post-transcriptional regulation (e.g., miR-23a-3p and miR-145-5p), post-translational modification (e.g., SIAH2-mediated ubiquitination), and substrate flux partitioning
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