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Upon increased stress, mitochondria can release their DNA through mitochondrial permeability transition pore opening (mPTP) [2], thus, to investigate this phenomenon, we specifically measured chronic ccf-mtDNA release

Additionally, pro-inflammatory cytokines such as TNF-, IL-1, and IFN- boost ROS production via mitochondrial and nicotinamide adenine dinucleotide phosphate oxidase pathways [68, 69]

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