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The ability of H3R inverse agonists/antagonists to suppress food intake is largely assumed to occur by removing the normal auto-inhibition of histaminergic neurons ( Figure 3 ), thereby increasing histamine levels and enhancing action at the anorexigenic H1R (78, 105, 115)

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Underpinnings of ASD: from Genetic to Environmental and Gene/Environment Etiologies The ongoing debate on the rise and prevalence of ASD over nearly half a century can be associated with the central scientific dogma that ASD is a highly heritable genetic disease of the brain

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