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Retatrutide's proposed mechanism of action involves GLP-1 receptor activation to slow gastric emptying and promote satiety, GIP receptor activation to support insulin secretion and adipose tissue regulation, and glucagon receptor agonism, which in trial settings has been associated with increased energy expenditure and lipolysis
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Retatrutide has trial data that looks promising but lacks the long-term, large-scale, real-world confirmation that only comes after FDA approval and widespread clinical use
These symptoms can lead to dehydration
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