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This HO-1-dependent cytoprotective mechanism may explain much of BPC-157s observed ability to protect cells against ischaemia-reperfusion injury and oxidative damage across multiple tissue types simultaneously

Ashley Koff, RD 00:18:18 So I think the dosing needs to be more personalized

Yuan J, Luo RZ, Fujii S, Wang L, Hu W, Andreeff M , Pan Y, Kadota M, Oshimura M, Sahin AA, Issa JP, Bast RC, Jr, Yu Y

As a result, FAO is suppressed, causing accumulation of intracellular long-chain fatty acids and reduced palmitate oxidation activity, ultimately enhancing VSMC proliferation and migration [158]

They are generally mild and self-limiting