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Western blot analysis of thoracic aorta proteins showed that the classic aging markers P21 and P16 were significantly downregulated following FOXO4-DRI treatment, whereas the expression levels of the cell proliferation marker Ki-67 and Lamin B, a key protein involved in nuclear structural stability, were markedly upregulated (Figure 1A)

S-glutathionylation enhances human cystathionine beta-synthase activity under oxidative stress conditions

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ChaC1 overexpression depletes GSH, initiating and executing ferroptosis

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