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Glial contributions to pathology can be primary (for example, astrocytic expression of mutant glial fibrillary acidic protein, GFAP, causing Alexander disease) or secondary, when glial cells react to pathology by mounting context-specific defensive responses
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Lino, M
Li Y, Wu H, Khardori R, Song YH, Lu YW, Geng YJ: Insulin-like growth factor-1 receptor activation prevents high glucose-induced mitochondrial dysfunction, cytochrome-c release and apoptosis
State 3, the maximal rate of ADP-dependent respiration [29], was examined with either pyruvate, palmitoylcarnitine (PC), or glutamate as oxidizable substrates
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