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Alterations in GSH metabolism and function have been implicated in various neurological aging while restoring redox balance and mitigating oxidative damage
HIF1-dependent glycolytic pathway orchestrates a metabolic checkpoint for the differentiation of TH17 and Treg cells
The present study found that 121 tomato UGT genes interacted with 12 different transcription factors, 90% of which were regulated by glutathione under CHT stress
As we age, stress, poor diet, and environmental toxins deplete glutathione levels, leading to increased inflammation, fatigue, and signs of aging
Integration of microbial metabolite signatures (e.g., volatile organic compounds, short-chain fatty acids, LPS derivatives) with host biomarkers can better capture the functional host-microbe interface
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