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However, a dysregulated increase in the level of these free radicals, as seen in mitochondrial diseases, can result in oxidative damage by overwhelming the cellular antioxidant defence mechanism, of which glutathione is the principal component [4]
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These measures help maintain the peptides suitability for research studies
Since the circadian melatonin cycle has been identified in the blood of every mammalian species where it has been examined (with the possible exception of some mice which genetically lack the enzymatic machinery to synthesize melatonin in the pineal gland), it was a reasonable assumption that the synchronizing actions of melatonin on the SCN is a result of the variation in day:night blood melatonin levels (44)
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