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4 Oxidative stress plays a central initiating role in the pathogenesis of vitiligo: the high metabolic activity of melanocytes leads to excessive generation of reactive oxygen species (ROS), while functional defects in the skins antioxidant system result in ROS accumulation, directly triggering mitochondrial dysfunction, endoplasmic reticulum stress, and apoptosis

The peptide stimulates GH release in a dose-dependent manner while maintaining the natural pulsatile pattern of GH secretion

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Downstream effects involve LC3-mediated autophagosome formation, G 2 /M phase arrest through cyclin regulation, and suppression of oxidative stress via ERK/AKT dephosphorylation