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Cysteine availability is typically the rate-limiting factor for synthesis, which is why N-acetyl cysteine (NAC), a cysteine donor, is one of the most effective strategies for raising glutathione levels [2][9]
Therefore, further investigation of the effect of TNF- on EBV reactivation and its specific mechanism is highly clinically important
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A signed consent was obtained from all included participants by the QBB, and WGS data used in the current study were obtained from the QGP as previously described 77
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