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Stroke models, as well as interventions to modulate neurogenesis after stroke (non-invasive such as running or EE, pharmaceutical or genetical) can have effects varying in both intensity and spatial distribution

[ii] Xu, C., Sun, L., Ren, F., Huang, P., Tian, Z., Cui, J., Zhang, W., Wang, S., Zhang, K., He, L., Zhang, W., Zhang, C., Hao, Q., Zhang, Y., Li, M., Li, W

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Its relevance comes not from an established clinical role, but from the way it has been reported to influence selected cellular processes commonly activated during tissue stress, injury-response signaling, and experimental repair models

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