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This suggests that acquisition of somatic mutations via DNA repair deficiency may not only be common in late disease (Fig
Change of hepatic insulin clearance was not different between groups (3.91 0.36 a.u to 3.68 0.4 a.u in R versus 3.39 0.17 a.u to 3.31 0.16 a.u in NR, P group over time = 0.49
[] Although TZP binds with comparable inclination to the GIPR as to native GIP, it is nevertheless five-folds higher when compared with the GLP-1 receptor
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