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Therefore, NDI1 can restore oxidative phosphorylation through functionally replacing CI, independently of time of day 16,27
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The metabolisation of each form is quite different and the effects differ considerably
Mirza et al
Monoamine Oxidase (MAO) Inhibition: Although its clinical relevance is debated, at high doses amphetamine acts as a weak competitive inhibitor of monoamine oxidase, the mitochondrial enzyme responsible for degrading free cytosolic monoamines
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