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The GSH-responsive intracellular re-assembly from nanoparticles to nanofibers can deplete existing intracellular GSH and achieve minimal effusion and the efficient accumulation of intracellular BSO, which may inhibit the biosynthesis of GSH and aggravate intracellular GSH depletion

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This shift toward MC4R selectivity makes PT141 a useful tool for isolating MC4Rmediated signalling including Gproteincoupled cAMP and phospholipaseC cascades, intracellular calcium flux, and ERK/MAPK pathway activation within neuronal and receptorexpressing model systems

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