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NNMT acts as a primary metabolic gatekeeper, predominantly active in fat tissue and liver cells, where it methylates nicotinamide (NAM) using S-adenosyl-L-methionine (SAM) as a methyl donor
Metabolites can be detected in urine
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Rohrig, F
[5] That finding should not be expanded into a universal claim that AOD9604 directly activates CPT1, increases mitochondrial number, induces oxidative gene expression, or increases fatty-acid oxidation in every tissue or model
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