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In humans, immunohistochemistry using a monoclonal antibody showed that the GLP-1 receptor protein is expressed in the pancreas, in smooth muscle cells of the kidney, lung, and gastrointestinal (GI) tract, and in myocytes of the heart [16]
In this regard, mice with a genetic deficiency in the klotho gene develop premature aortic stiffness that is associated with increased collagen deposition and reduced elastin in the medial layer of the aorta [186]
Conclusion Taken together, it presently appears that amongst individuals with HF, those most likely to benefit from GLP-1 RA treatment are people living with or at risk of developing obesity-related (with or without diabetes) HFpEF
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