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Similarly, the GCH1/BH4 pathway modulates ferroptosis through the production of tetrahydrobiopterin (BH4), a cofactor that selectively protects membrane phospholipids from peroxidation ( 3 Ferroptosis as a pathogenic amplifier in ischemic stroke 3.1 Mechanisms of ferroptosis in ischemic stroke Ferroptosis intensifies ischemic injury through mechanisms involving iron overload, lipid peroxidation, and mitochondrial dysfunction (Tuo and Lei, 2024) (Figure 2)

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In the canonical branch, IKK phosphorylates IB bound to p65/p50 heterodimers, targeting IB for proteasomal degradation