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Treatment with GLP-1RAs can reduce monocyte/macrophage accumulation in the arterial wall by inhibiting the adhesion of monocytes to activated endothelium from the arteries of C57BL/6 mice as well as in ApoE -/- mice [64]

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Plus another reason its so potent is because of the decreased binding of Long R3 IGF-1 to all known IGF binding proteins

GLP-1 products can be considered substances that modify body composition, which can result in a change in performance as opposed to strictly performance enhancing, he said