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A peer-reviewed study in the Journal of Endocrinology confirming that AOD9604 produces lipolysis and increases fat oxidation in obese mice via the beta-adrenergic pathway, with effects comparable to full-length HGH but without the insulin-disrupting side effects provided early mechanistic validation that informed the human trials
Additionally, independent scientists rely on it for exploratory metabolic investigations
The N-terminal tyrosine modification provides enhanced stability vs the unmodified HGH Fragment 176-191
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