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The TLR7 F506S mutation and other gain-of-function variants (F507S/L528I) show enhanced single-stranded RNA (ssRNA) binding affinity and agonist responsiveness, resulting in hyperactivated signaling that drives proinflammatory cytokine production in patient PBMCs [282, 283]
on the contrary, p-AMPK/AMPK, LXR, and ABCA1 protein levels were increased, and PARP-1 was decreased in T2DM + RYGB and T2DM + EX-4 groups when compared with T2DM group (Figure 6L)
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