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However, the molecular mechanisms responsible for the pathogenesis of COPD remain poorly understood
Statements Data availability statement The original contributions presented in the study are included in the article/ Supplementary Material
Analgesic activity in chronic pain models appears in the older literature, as do neuroprotective effects, effects on oxidative stress and on inflammation, and general neuromodulatory or neuromodulation framing
Under type 2 (T2) conditions (IL-13 stimulation), human airway epithelial cells (HAECs) generate oxidized phospholipids under control of the 15 lipoxygenase 1 (15LO1) enzyme and its binding with phosphatidylethanolamine-binding protein 1 (PEBP1) (6)
Brenner, BMC Chem
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